Elite Epithalon

89,99 

Elite Epithalon stimulates telomerase production, slowing aging, improving sleep, and enhancing immune function.

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Description

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Elite Epithalon is a cutting-edge peptide derived from epitalamine, a hormone secreted by the pineal gland. This peptide is renowned for its ability to stimulate the production of telomerase, an essential enzyme that plays a crucial role in maintaining telomere length. Telomeres, the protective end regions of DNA strands, shorten with each cell division, ultimately limiting the number of times a cell can replicate—an aspect closely linked to the aging process. By promoting telomere elongation, Elite Epithalon significantly enhances DNA replication capacity, thereby potentially decelerating aging and related health declines.

Mechanism of Action:
Elite Epithalon selectively interacts with specific DNA sites, facilitating epigenetic regulation of gene expression, which impacts oncogenes, telomerase, interleukin genes, and transcription factors. Furthermore, it influences protein synthesis relating to differentiation, proliferation, apoptosis, and telomere elongation in somatic cells. Research led by Dr. Khavinson demonstrated that cells treated with Epitalon can undergo ten more divisions than untreated controls. Not only does this extend cellular lifespan, but it also appears to uniquely contribute to increased life expectancy and decreased instances of cancer and overall mortality rates by regulating the endocrine, nervous, and immune systems.

Diurnal Cycle Regulation:
As part of the aging process, there is a notable decline in blood melatonin levels. Elite Epithalon plays a pivotal role in regulating biological rhythms and enhancing melatonin production via stimulation of the pineal gland. This modulation supports healthier sleep patterns and increases deep sleep, critical for bodily function and immune resilience.

Antioxidant Properties:
The peptide exhibits robust antioxidant activity, addressing the aging process by mitigating damage caused by reactive oxygen species (ROS). These cytotoxic agents can lead to DNA damage, cell death, or mutations that may promote cancer cell formation. Effective regulation of inflammation is paramount for overall health, as it can prevent or alleviate a spectrum of conditions, including rheumatoid arthritis and ulcerative colitis. Additionally, Elite Epithalon’s regulation of the endocrine system—boosting endogenous hormone levels—plays a significant role in maintaining overall health and well-being.

Composition of Elite Epithalon:
ARG-Epithalon: 500 mcg
D-mannitol: 200 mcg
Sodium Salcaprozate (SNAC): 2 mg

Dosage Instructions:
Take one capsule daily with a meal. This regimen is designed to maximize the peptide’s benefits while supporting overall health and vitality.

In summary, Elite Epithalon stands out as a promising supplement for those looking to manage the aging process actively, improve sleep quality, regulate hormonal levels, and enhance overall health through its multifaceted biological actions.

  1. Khavinson, V. K. et al. (2003). Peptide Bioregulation of Aging: Results and Prospects. Biogerontology, 4(4), 223–238. Link
  2. Anisimov, V. N. et al. (2011). Epithalon decelerates aging and suppresses development of breast adenocarcinomas in transgenic her-2/neu mice. Aging (Albany NY), 3(8), 788–797. Link
  3. Khavinson, V. K. et al. (2004). Epitalon and colon carcinogenesis in rats: Proliferative activity and apoptosis in colon tumors and mucosa. International Journal of Biomedical Science, 1(1), 56–61. Link
  4. Khavinson, V. K. et al. (2003). Effect of Epitalon on the lifespan increase in Drosophila melanogaster. Mechanisms of Ageing and Development, 124(1), 129–138. Link
  5. Khavinson, V. K. & Morozov, V. G. (2003). Peptides of pineal gland and thymus prolong human life. Neuro Endocrinology Letters, 24(3-4), 233–240. Link
  6. Naraikina, N. V., Anisimov, V. N., & Khavinson, V. K. (2012). Biomarkers of Aging: Replicative Senescence and Telomere Shortening. Aging and Health – A Systems Biology Perspective. Link
  7. Vinogradova, I.A., Bukalev, A.V., Zabezhinski, M.A., Semenchenko, A.V., Khavinson, V.K., An

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